Question

The breakdown of fatty acids via the Beta-oxidation metabolic pathway results in the production of (B),...

The breakdown of fatty acids via the Beta-oxidation metabolic pathway results in the production of (B), which feeds into the (?) metabolic pathway.

Homework Answers

Answer #1

Breakdown of fatty acids via beta oxidation occurs in 4 steps are as follows =

STEP 1 - the fatty acyl CoA is dehydrogenated with the FAD accepting the hydrogen atoms. FADH2 when oxidised in electron transport chain will produce 2 ATP molecules.

STEP 2 - this step forms a beta-hydroxy fatty acyl CoA.

STEP 3 - another dehydrogenation takes place with help of NADH, which when oxidized in electron transport chain will generate 2.5 ATPs.

STEP 4 - one molecule of acetyl CoA is liberated, leaving behind a fatty acid with 2 carbon atom less.

Summery of beta oxidation of fatty acids like palmittic acid (16 carbon). It undergoes 7 cycles, which give rise to 8 molecules of acetyl CoA.

Every molecule of acetyl CoA when oxidized in the TCA (tricarboxylic acid) cycle gives 10 molecules of ATP.

Know the answer?
Your Answer:

Post as a guest

Your Name:

What's your source?

Earn Coins

Coins can be redeemed for fabulous gifts.

Not the answer you're looking for?
Ask your own homework help question
Similar Questions
107. The breakdown of the fatty acid CH3CH2CH2CH2CH2CH2CH2CH2COOH via the β-oxidation pathway would:yield four moles acetyl...
107. The breakdown of the fatty acid CH3CH2CH2CH2CH2CH2CH2CH2COOH via the β-oxidation pathway would:yield four moles acetyl units only. True/False? 106.The breakdown of the fatty acid CH3CH2CH2CH2CH2CH2CH2CH2COOH via the β-oxidation pathway would:not occur unless the coenzyme A derivative was formed first.True/False? 105.Considering all forms of life, urea, ammonia and uric acid are major excretory forms of the a-amino groups of amino acids.True/False? 104.The removal of a-amino groups from amino acids for conversion to urea in animals may occur by transamination or...
The breakdown of the fatty acid CH3CH2CH2CH2CH2CH2CH2CH2COOH via the β-oxidation pathway would: yield three moles acetyl...
The breakdown of the fatty acid CH3CH2CH2CH2CH2CH2CH2CH2COOH via the β-oxidation pathway would: yield three moles acetyl units and one mol propionyl units. Group of answer choices True False Flag this Question Question 1111 pts During β-oxidation of long-chain fatty acyl CoA derivatives, energy is released. The following statements describe the way in which the energy is released and made available. Both NADH and FADH2 are produced which yield ATP via the electron transport chain. Group of answer choices True False...
Why do fatty acids bond with carnitine prior to beta-oxidation? a Carnitine is oxidized in beta-oxidation...
Why do fatty acids bond with carnitine prior to beta-oxidation? a Carnitine is oxidized in beta-oxidation reactions to generate acetyl-CoA and reducing power. b Fatty acyl-carnitine can be transported across the inner mitochondrial membrane while fatty acyl-CoA cannot. c Fatty acyl-carnitine is used in the beta-oxidation reactions.
17. If a hypothetical 10 carbon long fatty acid was metabolized by the beta oxidation pathway,...
17. If a hypothetical 10 carbon long fatty acid was metabolized by the beta oxidation pathway, how many cycles of the pathway would be required to completely break down the fatty acid to the end products of beta oxidation? A. 10 B. 1 C. 2 D. 4
2-Explain how beta-oxidation and fatty acids synthesis are reciprocally regulated
2-Explain how beta-oxidation and fatty acids synthesis are reciprocally regulated
Given that fatty acids are oxidized 2 carbons per cycle of beta oxidation, how are we...
Given that fatty acids are oxidized 2 carbons per cycle of beta oxidation, how are we able to oxidize a fatty acid with an odd number of carbons?
Acyl-CoA dehydrogenase deficiencies are diseases related to the impaired ability to oxidize fatty acids via beta...
Acyl-CoA dehydrogenase deficiencies are diseases related to the impaired ability to oxidize fatty acids via beta oxidation. Symptoms of these acyl-CoA dehydrogenase deficiencies include hypoketosis [low blood levels of ketone bodies], hypoglycemia [low blood glucose], and hyperammonemia [increased blood levels of ammonia] during long periods of fasting. Please explain why patients with acyl-coA dehydrogenase deficiencies have bouts of hypoketosis  [1- 2 sentences is sufficient]. (Please type answer, thanks!) (BioChem)
Which of the following statements about peroxisomes is FALSE? a. Beta-oxidation in peroxisomes can shorten very...
Which of the following statements about peroxisomes is FALSE? a. Beta-oxidation in peroxisomes can shorten very long fatty acids. b.Mammalian peroxisomes can synthesize some lipids, including bile salts. c.In plants, peroxisomes and glyoxysomes serve as the site of βετα− oxidation. d.Long chain fatty acids are transported into the peroxisome via a carnitine carrier protein where they are activated for oxidation.
A. In what organelle of the cell does β oxidation of fatty acids take place? chloroplasts...
A. In what organelle of the cell does β oxidation of fatty acids take place? chloroplasts cytosol nucleus mitochondria Golgi apparatus B. Which of the following forms of fatty acids can cross the inner mitochondrial membrane? free fatty acyl-carboxylate fatty acyl CoA fatty acyl-carnitine fatty acyl-glycerol
An important feature of the regulation of glucose catabolism via glycolysis and the pentose pathway is...
An important feature of the regulation of glucose catabolism via glycolysis and the pentose pathway is reflected in the observation that: A. transaldolase and transketolase are isomers of fructose-1,6-diphosphate aldolase and pyruvate ketolase, respectively. B. regulation is primarily achieved by segregation of glucose catabolism via the glycolytic pathway in the cytosol and the pentose pathway in the mitochondria. C. unlike the glycolytic pathway, the pentose pathway is activated uniquely by high levels of NADPH. D. the catabolism of glucose via...