Genome-wide association studies have identified many genomic loci at which the presence of one or more SNPs is associated with altered risk of a particular disease. Almost all of these SNPs have a very small influence on risk of the corresponding disease, typically with effect sizes of less than 1.5. Does this mean the affected genes are not likely to be good drug targets? If not, why not? What are the data so far on that?
Get Answers For Free
Most questions answered within 1 hours.