Question

question 4:

[b]: the [e] in the tunica media of your damaged blood vessels begins to contract, [f] blood flow to this portion of vessel, in an attempt to limit blood loss. Also, the [f1] of your tunica interna become "sticky" in an effort to help kick off the next step...

[c]: [g] circulating in your blood are attracted to exposed [h] and to the [f2] that became "sticky" at the end of step one. They begin to form a [i] and also begin to activate more cells of the same type to join them.

[d]: [q] factor (factor [1]) is released by damaged extravascular tissue. Together with the mineral [j], this factor activates factor [2], completing the [k] pathway. This factor activates factor [3], beginning the [l] pathway. Next, factor [4] is activated, then factor [5], known as [m]. This last factor initiates a [n] feedback cycle, activating more of itself as well as activating factor [6], known as [o1], a meshwork of which begins to form the actual [p1]. Finally, factor [7] is activated to strengthen the clot and begin its retraction. Blood cells of various sorts get caught in the [o2] mesh and help to solidify the [p2], stopping blood loss until the endothelial cells and rebuild the vessel lining.

Answers to plug inside bold letters or numbers:

• clot

• positive

• endothelial cells

• intrinsic

• platelet activation

• collagen fibers

• fibrin

• inflammation

• reducing

• extrinsic

• hemostasis

• coagulation

• tissue

• smooth muscle

• leukocytes

• vascular spasm

• calcium

• hemopoiesis

• thrombin

• common

• increasing

• magnesium

• thrombocytes

• negative

• plug

• I

• II

• III

• IV

• V

• VII

• IX

• X

• XI

• XIII

Answer 1

b- Vascular Spasm

e- smooth muscles

f- reducing

f1- endothelial cells

c- hemostasis

g- platelets

h- collagen fibres

f2- platelet activation

i- plug

d- Coagulation

q- tissue

j- Calcium

2- fibrin

k- common

3- factor VII

l- extrinsic

Dear Student, please specify exactly if any single doubt is present. Their are multiple pathways of intrinsic and extrinsic clotting pathways which cannot be guessed on so intricate fill in the blanks.

Due to time constraints, was able to solve this much only.