Question

How does hypertension effect preload and afterload of the heart?

How does hypertension effect preload and afterload of the heart?

Homework Answers

Know the answer?
Your Answer:

Post as a guest

Your Name:

What's your source?

Earn Coins

Coins can be redeemed for fabulous gifts.

Not the answer you're looking for?
Ask your own homework help question
Similar Questions
Match circulatory terms with the correct descriptions Refers to the myocardium's ability to stretch and contract...
Match circulatory terms with the correct descriptions Refers to the myocardium's ability to stretch and contract in response to filling of the heart with blood Answer Afterload Cardiac contractility Preload Stroke volume Causes stretch and increased pressure within the ventricles, which increases stroke volume Answer Afterload Cardiac contractility Preload Stroke volume This volume originates in the venous system Answer Afterload Cardiac contractility Preload Stroke volume The amount of blood within the ventricle that is ejected with each contraction Answer Afterload...
PROBLEM #2 INTRODUCTION: The contractile performance of heart muscle is determined by its inotropic state, preload...
PROBLEM #2 INTRODUCTION: The contractile performance of heart muscle is determined by its inotropic state, preload and afterload. These factors also determine the stroke output of the heart. The inotropic state is influenced by a variety of factors, including the extracellular Ca2+ concentration. PROBLEM #2: Predict the consequences (increase, decrease, no change) of the acute onset of hypocalcemia on the following:             CARDIAC MUSCLE                                                1. velocity of shortening             2. inotropic state (contractility)             3. peak force developed            ...
Which of the following would increase preload? a. decreased heart rate b. decreased plasma volume c....
Which of the following would increase preload? a. decreased heart rate b. decreased plasma volume c. decreased ventricular volume d. decreased venous return
1. Draw an x-y axis graph. On the x-axis, there is increasing preload; on the y-axis,...
1. Draw an x-y axis graph. On the x-axis, there is increasing preload; on the y-axis, is stroke volume. Graph the Frank-Starling curve: How would preload affect stroke volume? Draw this graph. Draw an x-y axis graph. On the x-axis, there is increasing afterload; on the y-axis, is stroke volume. How would increasing afterload affect stroke volume? Draw this graph. For # 3-5: Draw an x-y axis graph. On the x-axis, there is increasing muscle length; on the y-axis, there...
Discuss how long-term lung hypertension can affect the heart, causing a condition named cor pulmonale
Discuss how long-term lung hypertension can affect the heart, causing a condition named cor pulmonale
What effect does metoprolol have on heart rate response to exercise? Would you alter how you...
What effect does metoprolol have on heart rate response to exercise? Would you alter how you go about guiding exercise intensity for patients taking this drug?
how does hypertension impact capillary bulk flow?
how does hypertension impact capillary bulk flow?
How will cooling the heart down effect heart rate and contraction and state the physiological reasoning...
How will cooling the heart down effect heart rate and contraction and state the physiological reasoning behind this (good reasoning and explanation please).
A patient is given a large intravenous injection of norepinephrine that causes artery constriction, vein constriction,...
A patient is given a large intravenous injection of norepinephrine that causes artery constriction, vein constriction, and direct actions on the heart. Describe how this would affect heart afterload, preload, and contractility. Given all of these effects, do you expect that cardiac output will increase, decrease, or not change? Be sure to explain your answer.
A patient is given a large intravenous injection of norepinephrine that causes artery constriction, vein constriction,...
A patient is given a large intravenous injection of norepinephrine that causes artery constriction, vein constriction, and direct actions on the heart. Describe how this would affect heart afterload, preload, and contractility. Given all of these effects, do you expect that cardiac output will increase, decrease, or not change? Be sure to explain your answer.